No significant increases in uPAR were found in pretreatment determinations of patients with advanced small-cell lung cancer or non-small-cell lung cancer.

uPA and uPAR are involved in generation of drug-resistant small-cell lung cancer cell phenotype.

uPAR and beta1-integrin interactions are essential to signals induced by integrin matrix ligands or uPA in lung cancer cells.

role of single nucleotide polymorphism of uPA and uPAR in non-small cell lung cancer

High expression of uPA and ET-1 were found in lung adenocarcinomas.

The uPA expression was significantly correlated with TMPRSS4 expression in human lung and prostate cancers.

data on the stromal macrophages immunoreactivity of uPAR, MMP-2, and MMP-9 in a few small cell lung cancer (SCLC) and lung squamous cell carcinoma (SCC) biopsies was included. uPAR, MMP-2, and MMP-9 were confirmed in stromal cells including macrophages

High expressions of PLAU is associated with lung adenocarcinoma.