expression is closely related to invasiveness and metastasis in non-small-cell lung carcinoma

MTA1 may promote lung carcinogenesis by enhancing HIF-1alpha protein activity.

High metastasis-associated protein 1 nuclear expression is associated with non-small cell lung cancer.

Mta1 protein overexpression is an independent prognostic factor for patients with non-small-cell lung cancer.

Overexpression of metastasis-associated protein 1 is significantly correlated with tumor angiogenesis in patients with early-stage non-small cell lung cancer.

MTA1may play an important role in the smoked-related progress of non-small cell lung cancer

MTA1 functions in regulating the invasive phenotype of lung cancer cells and this regulation may be through altered miRNA expression

MTA1 plays a critical role in regulating the malignant behaviors of small cell lung cancer

We revealed a new molecular mechanism of MTA1-mediated invasion and metastasis in lung cancer through downstream target EpCAM, and interfering with EpCAM function may be a novel therapeutic strategy for treatment of MTA1-overexpressing lung carcinoma

Metastasis-associated protein-1 expression levels in HPV-infected non-small cell lung cancer tissues correlated positively with tumor stage and nodal metastasis.

High expressions of MTA1 is associated with epithelial to mesenchymal transition and metastasis in non-small-cell lung cancer.

our studies indicate that Curcumin increases the sensitivity of Paclitaxel-resistant non-small-cell lung cancer cells to Paclitaxel through microRNA-30c-mediated MTA1 reduction. Curcumin might be a potential adjuvant for non-small-cell lung cancer patients during Paclitaxel treatment.

Overexpression of MTA1 could be a marker of poor prognosis in Chinese NSCLC patients, but not in lung cancer or small cell lung cancer.

this study shows relationship between MTA1 and spread through air space and their joint influence on prognosis of patients with stage I-III lung adenocarcinoma