Involvement of S100A2 and S100A4 in the progression of lung adenocarcinomas provides a list of targets regulated by S100A2 and S100A4.

high S100A4 expression is related to a poor prognosis through hematogenous metastasis in pulmonary adenocarcinoma.

expression of S100A4 protein and loss of E-cadherin protein expression are significantly associated with tumor progression in non-small cell lung cancer.

staining of S100A4 was significantly associated with patients' poor prognosis in lung squamous cell carcinoma but not lung adenocarcinoma

these results suggest that S100A4 plays an important role in lung carcinogenesis by means of cell proliferation and motility by a pathway similar to that in pancreatic cancer.

S100A4 promotes lung tumor development through inhibiting autophagy in a beta-catenin signaling and S100A4 receptor RAGE-dependent manner.

findings uncover a novel S100A4 function and highlight its importance in controlling NDUFS2 expression to regulate the plasticity of mitochondrial metabolism and thereby promote the invasive and metastatic capacity in lung cancer

Clinicopathological and prognostic value of S100A4 expression in non-small cell lung cancer: a meta-analysis.