miR-495 regulates the multi-drug resistance by modulation of ATP7A expression in non-small cell lung cancer and miR-495 may serve as a potential biomarker for the treatment of multi-drug resistant non-small cell lung cancer patients with high ATP7A levels

TSPAN12 promotes chemoresistance and proliferation of small cell lung carcinoma under the regulation of miR-495.

mir495 is down-regulated in lung tumor samples. mir495 interacts with 3'-UTR of GRP78.

MicroRNA-495 inhibits the progression of non-small-cell lung cancer by targeting TCF4 and inactivating Wnt/beta-catenin pathway

the findings of the present study demonstrated that miR495 was downregulated in nonsmall cell lung cancer tissues and cells. In addition, miR495 suppressed the proliferation of lung cancer cells by directly targeting HMGA2

Deregulation of UCA1 expression may be involved in the development of chemoresistance to cisplatin in the treatment of non-small-cell lung cancer via regulating the signaling pathway of microRNA-495/NRF2.