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Cancer metastasis database

Annotation category 5

Literatures

Literature evidence

Links to all GeneRIF Items

5329

PubMed (Gene Name)

Literature evidence

12174885 (PLAUR)

circulating UPA is not up-regulated by the circulating P105 fraction of the HER-2/neu proto-oncogene: in vivo evidence from patients with advanced non-small cell lung cancer

12704669 (PLAUR)

uPAR expression in lung cancer is regulated at a posttranscriptional level by uPA

14507113 (PLAUR)

Significant increases in uPAR were found in pretreatment determinations of patients with advanced small-cell lung cancer or non-small-cell lung cancer.

17327908 (PLAUR)

uPA and uPAR are involved in generation of drug-resistant small-cell lung cancer cell phenotype. uPAR-positive cells may define a functionally important population of cancer cells in SCLC.

17548471 (PLAUR)

These observations demonstrate a novel regulatory role for p53 as a uPAR mRNA binding protein that down-regulates uPAR expression, destabilizes uPAR mRNA, and thereby contributes to the viability of human airway epithelial or lung carcinoma cells.

17706320 (PLAUR)

High tumour cell C4.4A expression is associated with shorter survival for non-small cell lung cancer patients.

18940913 (PLAUR)

uPAR and beta1-integrin interactions are essential to signals induced by integrin matrix ligands or uPA in lung cancer cells.

20937265 (PLAUR)

role of single nucleotide polymorphism of uPA and uPAR in non-small cell lung cancer

23030781 (PLAUR)

Multivariate survival analysis demonstrated that high levels of uPAR(I) were significantly associated with short overall survival in small cell lung cancer patients

25120753 (PLAUR)

these data suggest that uPAR and CXCR4 co-expression predicts worse prognosis of small cell lung cancer patients

26050228 (PLAUR)

data on the stromal macrophages immunoreactivity of uPAR, MMP-2, and MMP-9 in a few small cell lung cancer (SCLC) and lung squamous cell carcinoma (SCC) biopsies was included. uPAR, MMP-2, and MMP-9 were confirmed in stromal cells including macrophages

29961070 (PLAUR)

results demonstrated that PLAUR induces geftinib-resistance through EGFR/p-AKT/survivin signaling pathway in gefitinib-resistant human lung adenocarcinoma cells. PLAUR could be a novel therapeutic target for gefitinib-resistant NSCLC patients.

30280780 (PLAUR)

The epigenetic regulation of uPAR by HDAC1 confirms its indirect role in regulating p38 MAPK as lung tumors progress.