these data support the involvement of miR-22, miR-24, and miR-34a in advanced non-small cell lung cancer

findings support a lung metastasis-promoting function of the miR-23b/27b/24 cluster of miRNAs, which functions in part through the direct inhibition of PSAP in breast cancer

MiR-24 acted as an oncomir, at least partially through regulation of its functional target NAIF1 in non-small cell lung cancer.

Data found that both miR-7a and miR-24-3p had a significantly higher copy number in non-malignant lung tissues (NMLTs) samples compared to lung adenocarcinoma (AC) tissues and that they both modulate the SOX18 transcript in AC cells.

Study found miR24 to be associated with menin, affecting the activity of the SMAD3 pathway in lung cancer by inhibiting menin expression. miR24 may promote the growth and metastasis and inhibit the apoptosis of lung cancer cells by targeting menin.

these findings define a novel insight into the miR-24-3p/FGFR3 signaling axis in regulating lung adenocarcinoma progression

We found that miR-24 promoted lung carcinoma development by increasing cell proliferation and inhibiting cell apoptosis via targeting MAPK7