MiR-210 promotes a hypoxic phenotype and increases radioresistance in human lung cancer cell lines.

Higher serum miR-210 levels were significantly correlated with the clinical stage and the presence of regional lymph node metastasis in patients with nonsmall-cell lung cancer.

co-expression in cancer and stromal cells is an independent prognosticator for disease-free survival in non-small cell lung cancer

Study shows that an increase of both exogenous and endogenous TIMP-1 led to the upregulation of miR-210 in a CD63/PI3K/AKT/HIF-1-dependent pathway in lung adenocarcinoma cells.

miR-210 may act as a potential tumor oncogene in lung adenocarcinomas.

microRNA-210 has moderate diagnostic value for lung cancer [review and meta-analysis]

MiR-210 expression was elevated and overexpression indicated poor progression-free survival and expression level of miR-210 in serum and sputum showed significant diagnostic value for non-small cell lung cancer.

Our results suggested that miR-210 plays a great role in autophagy of lung cancer cell by targeting ATG7.

we confirmed that LOXL4 could serve as a downstream target of miR-210 and miR-210 promoted lung cancer progression via targeting LOXL4. In A549 and H1650 cells, knockdown of LOXL4 dramatically repressed lung cancer cell proliferation, migration, and invasion.

High miR210 expression is associated with metastasis of lung cancer.

lung CSC-derived exosomal miR-210-3p contributes to a pro-metastatic phenotype in lung cancer by targeting FGFRL1.

tumor promoting effects of exosomal microRNA-210 derived from lung cancer cells on lung cancer through the RUNX3/PI3K/AKT signaling pathway axis.