expression of miR-203 is downregulated in lung cancer cells; miR-203 negatively regulates survivin protein expression and inhibits the proliferation and invasion of lung cancer cells

miR-203 inhibits cell proliferation and migration of lung cancer cells by targeting PKCalpha.

Repression of SRC by miR-203 suppressed the proliferation and migration and promoted the apoptosis of lung cancer cells.

Results showed that miR-203a downregulation induced ERGIC3 overexpression in non-small cell lung cancer cells.

Downexpression of MiR-203 is associated with non-small cell lung cancer.

LASP-1, mediated by miR-203, has crucial functions in the proliferation, migration and invasion of human non-small cell lung cancer

results highlight a feedback loop between miR-203 and ZEB2, which participates in the pathogenesis of lung adenocarcinoma

Results found miR-203 and DKK1 to be expressed at differential levels in cisplatin-sensitive and -insensitive lung adenocarcinoma tissues; there was also a negative correlation between them.

Taken together, the results suggested that expression of miR-203 inhibited non-small-cell lung cancer tumor growth and metastasis by targeting RGS17

HCP5 is a SMAD3-responsive long non-coding RNA that promotes lung adenocarcinoma metastasis via miR-203/SNAI axis.

LncRNA LINC00342 regulated cell growth and metastasis in non-small cell lung cancer via targeting miR-203a-3p.

Long noncoding RNA FBXL19-AS1 induces tumor growth and metastasis by sponging miR-203a-3p in lung adenocarcinoma.

Mechanism of epithelialmesenchymal transition inhibited by miR203 in nonsmall cell lung cancer.

Exosomal HOTAIR promotes proliferation, migration and invasion of lung cancer by sponging miR-203.

Low miR203 expression is associated with proliferation and metastasis of non-small-cell lung cancer.

Clinical significance and functions of miR-203a-3p/AVL9 axis in human non-small-cell lung cancer.