Our results suggest that down-regulation of miR-200b could lead to E2F3 overexpression and in turn contribute to chemoresistance of lung adenocarcinoma cells to docetaxel.

Overexpression of miR-200b led to the downregulation of ZEB2 at protein level so our results suggested that ZEB2 regulated by miR-200b may contribute to multi-drug resistance of small cell lung cancer.

novel HDAC1/4/miR-200b/E2F3 signaling contributes to chemoresistance of human lung adenocarcinoma cells

Underexpression of MIR200B was associated with lung adenocarcinoma.

The miR-200b and the miR-183~96~182 cluster target Foxf2 to inhibit invasion and metastasis in lung cancers.

Increased miR-200 expression may contribute to early lung tumorigenesis

miR-200b regulates autophagy associated with chemoresistance in human lung adenocarcinoma.

miR-200b and ZEB1 might have roles in sensitivity to nintedanib in non-small cell lung cancer cells

miR200b and miR200c targeted the expression of RhoE and inhibited the malignancy of nonsmall cell lung cancer (NSCLC) cells, and the downregulation of miR200b and miR200c may contribute to the high expression of RhoE in NSCLC.

Results find miR-200b dysregulated in both triple negative (TNBC) and double positive breast cancer. Its expression level was correlated with lung and bone metastatic genes. miR-200b presents a great potential for the future advancements in the diagnostic/prognostic of TNBC.

High miR200 expression is associated with metastasis in lung Adenocarcinoma.

microRNA-200b-3p is upregulated in lung adenocarcinoma cells and associated with cell proliferation and metastasis. MicroRNA-200b-3p promoted lung adenocarcinoma cell proliferation and metastasis by suppressing adenosine triphosphate-binding cassette transporter A-1.