These findings suggest that miR-150 and miR-3940-5p interfere in non-small cell lung carcinoma tumorigenesis and progression, and in the direct or indirect pathways of miR-150 and miR-3940-5p to affect p53 expression.

Findings indicate the role of miR-150 as an oncogene in lung cancer through the inhibition of SRC kinase signalling inhibitor 1 (SRCIN1) translation.

data highlight a novel molecular interaction between miR-150 and BAK1 and provide a novel strategy for non-small cell lung cancer therapy via the down-regulation of miR-150 expression

Our data indicated that overexpression of miR-150 in non-small cell lung cancer tissues has prognostic value.

The results suggest that miR-486 and miR-150 could be potential blood-based biomarkers for early diagnosis of non-small cell lung cancer.

Study demonstrates that JMJD2A contributes to tumorigenesis in non-small cell lung cancer (NSCLC) by regulating miR-150. Additionally, JMJD2A overexpression is associated with a poor prognosis for NSCLC patients.

miR-29a-3p and miR-150-5p were identified as circulating biomarkers that correlated with delivered thoracic radiation dose in non-small cell lung cancer

The analysis of tumor and matched normal lung tissues indicates that miR-150 downregulation in lung tumors correlates with higher CDK3 levels. In addition, miR-150 transfection experiments with cancer-derived cell lines reveal that miR-150-mediated CDK3 suppression directly induces growth inhibition.

Study suggest that the downregulation of miR1505p and the overexpression of MMP14 may be deeply involved in the pathogenesis of lung squamous cell carcinoma.

MiR150 plays an important role in the development of lung cancer by serving as an oncogene via the SIRT2/JMJD2A signaling pathway.

C-myc/miR-150/EPG5 axis mediated dysfunction of autophagy promotes development of non-small cell lung cancer.