miR-144 promotes the apoptosis of lung cancer cells, and inhibits the proliferation, invasion and migration of lung cancer by regulating ZEB1 gene.

that miR-144 could reverse the function of bladder cancer associated transcript 1 on nonsmall cell lung cancer cells' phenotype

Study verified that miR-144 was downregulated in lung adenocarcinoma (LUAD) tissues, which was functionally associated with the propagation and invasiveness. These results suggested that miR-144 may be one of the candidates for new molecular diagnosis and prognosis in LUAD.

In lung squamous cell carcinoma (LUSQ) cells, 13 and 15 genes were identified as possible oncogenic targets that might be regulated by miR-144-5p and miR-144-3p, respectively.

Study found that miR1443p could regulate the cisplatin resistance of lung cancer cells via Nrf2.

interference of LINC00483 weakens its competitive binding ability to miR-144, thus reducing HOXA10 expression, and enhancing radiosensitivity in lung adenocarcinoma.

MIRN144-3p was lowly expressed in non-small cell lung cancer.

LINC00466 could restrain the miR-144 expression to up-regulate HOXA10 and, therefore, promote lung adenocarcinoma.