data thus indicating the potential of miR-21, miR-143 and miR-181a as a novel diagnostic or prognostic biomarker for non small cell lung cancer.

The up-regulated miR-143 in lung cancer could significantly inhibit cell migration and invasion, and this might work through targeting CD44v3

miR-143 inhibits cell proliferation by targeting autophagy-related 2B in non-small cell lung cancer H1299 cells.

The relative expression of miR-143-3p was significantly upregulated in relapsed lung adenocarcinoma samples using FFPE tissue, which showed possible clinical utility as biomarkers predicting recurrence after curative surgery.

Long non-coding RNA UCA1 inhibits MAPK1 expression by miR-143 up-regulation, modulating the proliferation, migration and invasion of human lung cancer cells.

miR-143 rs3733845 polymorphism is associated with an increased lung adenocarcinoma risk among nonsmoking females residing in northeast China.

miR-143-5p and its target genes were involved in the molecular pathogenesis of lung adenocarcinoma.

CL-pVAX-miR-143 might be a promising strategy for clinical treatment of non-small cell lung cancer.

we identified that miR-143-3p was involved in the BM of lung cancer cell via down regulation of VASH1.