lung cancer gene and literature database Home
Cancer metastasis database

Annotation category 5

Literatures

Literature evidence

Links to all GeneRIF Items

355

PubMed (Gene Name)

Literature evidence

12037669 (FAS)

alteration associated with nodal metastasis in non-small cell lung cancer

12824926 (FAS)

increased expression of soluble Fas in pleural effusion associated with lung cancer

14512182 (FAS)

Fas polymorphism is associated with altered apoptotic capacity in lymphocyte cultures, and risk of lung cancer

15937082 (FAS)

FAS polymorphisms contribute to susceptibility of lung cancer.

17014925 (FAS)

These results suggest that the FAS-1377G>A and -670A>G and FASL-844C>T polymorphisms do not significantly affect the susceptibility to lung cancer in Koreans.

17708363 (FAS)

There was no relationship between Fas-1377 G-->A polymorphism and lung cancer

19240174 (FAS)

Polymorphisms in the FAS is associated with early stage non-small cell lung cancer.

21430781 (FAS)

knockdown of FAS and several components of the NF-kappaB pathway specifically enhanced cell death induced by the EGFR TKI erlotinib in EGFR-mutant lung cancer cells

21466843 (FAS)

Celastrol can induce apoptosis of human non-small-cell lung cancer A549 cells through activation of both mitochondria- and Fas/FasL-mediated pathways.

22821704 (FAS)

Non-small-cell lung cancer patients carrying XRCC1 Arg194Arg, FAS-1377GG, and FASL-844T allele displayed completely no response to platinum based drugs.

25596086 (FAS)

FasL -844 SNP is associated with the susceptibility of pulmonary AD in age <60 years people. Fas -1377 SNP may modify the association of FasL -844 SNP with the risk of pulmonary adenocarcinoma.

26823709 (FAS)

Suggest reduced membranous Fas expression as a mechanism of apoptotic resistance in non-small cell lung carcinomas.

28320962 (FAS)

Study identify genes highly expressed in Kras knockout lung cancer cells, including the Fas receptor gene. Antibodies that activate Fas receptor selectively induced apoptosis in Kras-independent lung cancer cells suggesting that FAS is involved in KRAS-related drug resistance.

17951400 (Fas)

Fas(-) osteosarcoma cells are selected during lung metastases formation and inhibition of Fas signaling in tumors or lack of FasL in the host environment allows the proliferation of Fas(+) osteosarcoma cells in the lungs and promotes metastases growth.

19265159 (Fas)

Fas signal can promote lung cancer growth in vivo by recruiting myeloid-derived immunosuppressive cells and T regulatory cells into the tumor microenvironment via inflammatory prostaglandin E2.