High NEAT1 expression is associated with metastasis in lung cancer.

Study showed that NEAT1 could function as a competing endogenous lncRNA in lung cancer, mediating CTR1 by sponging hsa-mir-98-5p.

NEAT1 modulated the expression of E2F3 and MiR377 and promoted non small cell lung cancer progression.

NEAT1 expression is controlled by Oct4 transcriptional regulation in lung cancer.

NEAT1/hsa-mir-98-5p/MAPK6 is involved in the development and progress in non-small-cell lung cancer.

Study confirmed that galectin-3 as a ligand of TLR4 induced TLR4 signaling activation in lung adenocarcinoma cells, thereby activating downstream p65 nucleus translocation, promoting NEAT1 expression, and finally affecting lung adenocarcinoma cell proliferation and migration.

NEAT1 regulates lung cancer cell progression by competing endogenous RNA network of NEAT1/let-7a/IGF2.

it was exhibited that Wnt signal pathway was inactivated by knockdown of NEAT1 whereas activated by NEAT1 overexpression in A549/CDDP cells; it was indicated that NEAT1 could exert a novel biological role in non-small cell lung cancer chemo-resistance

NEAT1 mediates paclitaxel-resistance of non-small cell of lung cancer through activation of Akt/mTOR signalling pathway.

NEAT1 up-regulation induced by HIF-2alpha over-expression could promote the progression of non-small cell lung cancer under hypoxic condition.

LncRNA NEAT1/miR-1224/KLF3 contributes to cell proliferation, apoptosis and invasion in lung cancer.

LncRNA NEAT1 SNP rs2239895 is associated with the risk of lung squamous cell carcinoma in a Chinese northeast Han population.

EGCG regulates CTR1 expression through its pro-oxidative property in non-small-cell lung cancer cells.

LncRNA NEAT1 regulated cell proliferation, invasion, migration and apoptosis by targeting has-miR-376b-3p/SULF1 axis in non-small cell lung cancer.

Association between NEAT1 polymorphism and the risk of lung cancer: A protocol for systematic review and meta-analysis.